Androgenic alopecia can be accelerated by TRT and AAS, particularly in genetically susceptible men. The driving mechanism is DHT (dihydrotestosterone) acting on follicular androgen receptors, not testosterone itself.
Elevated DHT from testosterone conversion via 5-alpha reductase — especially on supraphysiologic doses
High free testosterone providing more substrate for DHT conversion at follicle level
Use of high-androgenicity compounds (trenbolone, masteron, DHT derivatives) that directly activate follicular receptors
Genetic predisposition to follicular androgen sensitivity — bloodwork cannot predict this
GearCheck correlates your symptoms with your actual lab values and flags the specific markers driving the issue — not just a generic panel review.
Analyze My Bloodwork →GearCheck provides blood marker analysis and harm reduction education. Information is for educational purposes only and does not constitute medical advice. Always consult a healthcare professional.